<p>Often in a concussion, the initial impact will lead the brain to release the excitatory neurotransmitter glutamate at the areas of damage, which causes a rapid depolarization of the local neurons. After this, the axonal sodium/potassium pump goes into overdrive to rebalance ion/voltage imbalance, more ATP (energy) is required, there is a jump in glucose metabolism, and extracellular swelling starts, reducing blood flow. Then the increase in metabolism with reduced blood flow causes an energy crisis in the cell. Cell is unable to carry out regular activity due to lack of energy and often entire cell groups will go dormant to preserve themselves. This causes a low voltage EEG. However, for the record, a low voltage EEG is also a normal phenotype (Johnstone et al, 2005).&nbsp;</p><p>It is very likely that, after a career of having hard knocks to the skull, that clients’ brains will form scar tissue at the sites of the accident (after initially experiencing a state of inflammation). If the inflammation is not able to properly heal the brain—which is its’ function—then the brain will ultimately learn to handle the inflammation by altering cellular structures to form tougher tissues, which would consequentially prevent blood perfusion at the sites of the injury and prevent the slower brainwaves of delta and theta from being read from the surface.</p><p><br></p>