<b>Stimulants</b> <br><br><img src="http://via.placeholder.com/600x260" alt="" width="600" height="260" role="presentation" class="img-responsive atto_image_button_text-bottom">&nbsp;<br><br><ul><ul><li>
Used for ADHD disorders mainly</li>
<li>ADHD profiles:
<ul><li>High theta/beta ratio or theta/alpha ratio&nbsp;</li><li>Higher relative theta&nbsp;</li><li>Frontal spindling beta&nbsp;</li><li>Mu rhythm&nbsp;</li><li>Temporal lobe alpha</li>
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Effects
</b><ul><ul><li>Normalization of EEG</li><li>No change from baseline</li><li>Decrease in theta, theta/alpha, and theta/beta ratios</li><li>Increase in beta amplitudes</li></ul>


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Stimulants increase the activity in the RAS, with the Raphe nucleus releasing norepinephrine, decreasing the polarization in the reticular nucleus of the thalamus and thus increasing the “clocking” or peak frequency of the rhythmic alpha activity and increasing faster activity.
<br><br><b>Amphetamines:</b> Both dextro and methamphetamines like Dextrostat or Adderal are similar in effect, with decreased slower activity and increased beta from 12-26 Hz. There is a paradoxical increase in alpha noted in the CEEG work of Itil (Itil et al., 1980). This is likely from the increased activation effect mentioned in the opening section.
<br><br><b>Methylphenidate:</b> Ritalin produces a decrease in delta and theta, with a more pronounced posterior alpha increase and an increase in low beta, with effects delayed up to 6 hours, compared to the rapid effects of the amphetamines.